selected publications
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academic article
- Oestrogen receptor ß mediates the actions of bisphenol-A on ion channel expression in mouse pancreatic beta cells. DIABETOLOGIA. 1667-1680. 2019
- Pancreatic alpha-cell mass in the early-onset and advanced stage of a mouse model of experimental autoimmune diabetes. SCIENTIFIC REPORTS. 1-12. 2019
- DEXI, a candidate gene for type 1 diabetes, modulates rat and human pancreatic beta cell inflammation via regulation of the type I IFN/STAT signalling pathway. DIABETOLOGIA. 1-14. 2018
- IFN-alpha induces a preferential long-lasting expression of MHC class I in human pancreatic beta cells.. DIABETOLOGIA. 636-640. 2018
- Mitochondria as target of endocrine-disrupting chemicals: implications for type 2 diabetes. JOURNAL OF ENDOCRINOLOGY. 27-45. 2018
- PDL1 is expressed in the islets of people with type 1 diabetes and is up-regulated by interferons-¿ and-¿ via IRF1 induction.. EBIOMEDICINE. 367-373. 2018
- Timing of Exposure and Bisphenol-A: Implications for Diabetes Development. FRONTIERS IN ENDOCRINOLOGY. 1-10. 2018
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congress communication
- BCL-XL, but not complement C3, protect alpha-cells against metabolic stress- or proinflammatory-induced apoptosis 2019
- BCL-XL, but not complement C3, protects alpha-cells against metabolic stress- or proinflammatory-induced apoptosis 2019
- BISPHENOL-A MODULATES ION CHANNEL EXPRESSION AND FUNCTION VIA ESTROGEN RECEPTOR ¿ IN MOUSE PANCREATIC BETA CELLS 2019
- Bisphenol-A modulates ion channel expression and activity in mouse pancreatic beta cell via oestrogen receptor beta (ERbeta) 2019
- Differential effects of chronic exposure to bisphenol A on ion channel activity and expression in mouse pancreatic ß-cells 2019
- Exposure to bisphenol-A induces pancreatic ß-cell apoptosis 2019
- Interfering with beta-cells or: how type I interferons are implicated in type 1 diabetes 2019
- Interfering with ß-cells or: how type I interferons are implicated in type 1 diabetes 2019
- xBCL-XL, but not complement C3, protects alpha cells against metabolic stress- or proinflammatory-induced apoptosis 2019